Pregnancy and the thyroid gland

Pregnancy is an important impact for the thyroid, even if the future mother is a healthy woman and has never had thyroid problems. The reason is that the thyroid gland must increase its production of thyroxine by 50 percent and is forced to work hard to cope with pregnancy. It is estimated that the

Pregnancy is an important impact for the thyroid, even if the future mother is a healthy woman and has never had thyroid problems. The reason is that the thyroid gland must increase its production of thyroxine by 50 percent and is forced to work hard to cope with pregnancy.

It is estimated that the pregnant woman's thyroid produces between 30 and 50 percent more thyroxine than under normal conditions. To maintain the level of T4-Free stabilized blood has to increase its rate of production, since the mother also transfers to the fetus a portion of their hormones, enough to maintain normal development, if the fetus had problems with his thyroid. Here you will find very valuable information about pregnancy and the thyroid gland.

What is the relationship between pregnancy and the thyroid gland

The important stimulus of the thyroid in pregnant women is due to the influence of a number of specific factors: La 1. Chorionic gonadotropin.

It is the pregnancy hormone par excellence and is produced in the placenta. It is detected in the mother's blood or urine and is the basis of the pregnancy test. It influences the stimulation of the thyroid, especially during the first trimester. Its production begins immediately after conception, after 2 or 3 days and the effects in the body of the pregnant woman are very similar to TSH. Chorionic gonadotropin acts as a stimulant for the thyroid. Approximately, 18 percent of pregnant women have a slight thyroid stimulus during the first trimester that goes unnoticed among the other discomforts that the pregnant woman feels.During the first trimester, there may be a slight increase in T4 and a stagnation of TSH, which is normal and should not be confused with hyperthyroidism. In the knownfalse hyperthyroidism of the first trimester of pregnancy,

there is an increase of thyroid hormones in blood and an inhibition of TSH, antithyroid antibodies are normal and, above all, ultrasound is normal. In the second and third trimesters chorionic gonadotropin levels fall and the picture tends to remit. Inauthentic hyperthyroidism hay, there is an increase in the vascularization in the thyroid seen in the eco-Doppler-color study. In false hyperthyroidism, thyroid ultrasound is normal.

2. Estrogens. In the second and third trimesterthere are also factors that modify thyroid function. The female hormones themselves, mainly estrogen, could be involved. Thyroid hormones circulate in the blood together with a protein called TBG (Thyroxin Bindig Globulin or Thyroxine Transporting Globulin or Thyroxine Transporter), and in pregnancy, TBG levels skyrocket. Between 16 and 20 weeks of gestation, the TBG figure in the blood doubles. In these circumstances, the T4 and T3 blood levels are altered and may give the false impression of a functional alteration, but if the T4-Free is not modified and remains normal there is no hypothyroidism in pregnancy.

As you can see, the pregnancy and the thyroid gland are related. In pregnancy, the mother's thyroid may grow a little. This growth is registered by ultrasound and only occurs in 10-15 percent of cases. This discrete increase in size returns to its original state after delivery. Marisol Nuevo.Guiainfantil.com